development of IAA was thought to be likely due to oxidase deamination of NMT (Barker et al., 1980), but was later on proven for being also in part by immediate deamination by MAO (Barker et al., 1982). Pretreatment Together with the MAO-I iproniazid in rat complete brain homogenates inhibited IAA development by 83%, NMT and DMT-NO formation were b
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